Although these findings have been disputed (Wang et al., 2014), and more work will be required before conclusions regarding the neurogenic capacity of the striatum are reached, an examination of the effects of depression and ADT on DCX expression and neuroblast number in this region may help elucidate whether our findings reflect a general impairment of migration among SVZ-derived neuroblasts. The gene discussed is DCX; the disease is depressive symptom measurement.