Bronchial epithelial cells also express the receptor for IL-4 (IL-4RA), and the activation of these cells by IL-4 induces, among other inflammatory parameters [15], the production of chemokines, for example, CCL2, CXCL-8, among others [7, 13, 14, 16, 17], which modulate leukocyte traffic and consequently airway inflammation in asthma. This evidence concerns the gene IL4 and asthma.