EGFR and colonic neoplasm: An overexpression of miR-147 in CRC cell lines could induce MET by increasing the expression of CDH1 and decreasing that of ZEB1, inhibit the invasion and motility of the cells, and cause a G1 arrest by upregulating p27 and downregulating cyclin D1, as well as reverse the chemoresistance of the colon cancer cell line HCT116 to EGFR inhibitor, gefitinib [32].