Given that MYOM3 is predominantly expressed in slow and intermediate speed (type I and IIa) skeletal fibres (29) which are less affected in DMD relative to fast myofibres (40), it is possible that the difference in the kinetics of these biomarkers is partially due to the differential sensitivity of these muscle fibre types to exercise-induced damage. Here, MYOM3 is linked to Duchenne muscular dystrophy.