On one hand, inhibition of PP2A is believed to have tumor-promoting function [5] through induction of phosphorylation and activation of several substrate kinases, including IKK (IκB kinase), JNK (c-Jun N-terminal kinase), ERK (extracellular signal-related kinase), p38, Akt, and PKC (protein kinase C), most of which accelerate growth. This evidence concerns the gene PTPA and neoplasm.