KLF4 and neoplasm: We previously reported that RAS-ERK signaling, a prosurvival pathway, is maintained in TNBC cells by KLF4, in part through its regulation of miR-206.37 In contrast to the prominent effect of miR-206 on tumor initiation and cell survival, on its own this miR has only limited effects on ERK activity.37 We therefore sought to better understand how endogenous miR-206 can promote anoikis resistance.