Furthermore, we also measured expression of factors that are deregulated in CLL but that are not known to be directly involved in the pathogenesis of CLL (including Ataxin (ATXN1) [6, 7], FCER2/CD23 [6, 7], early B-cell factor 1 (EBF1) [7], myristoylated alanine-rich protein kinase C substrate (MARCKS) [8] and Ly9/CD229 [56]. Here, MARCKS is linked to B-cell chronic lymphocytic leukemia.