Acute settings as in institutionalized patients [18] or following falls and fractures [6, 34] have reported a higher incidence of SIADH which could be related to higher exposure to hypotonic fluids unmasking the disease [18] or due to the confounding influence of pain or opioid medications in trauma settings, which are potent stimulus for ADH release [38]. This evidence concerns the gene AVP and inappropriate ADH syndrome.