Analysis of additional markers involved in ATII-to-ATI cell trans-differentiation, such as forkhead box M1 (FoxM1), a transcription factor described to be essential for ATII-to-ATI cell trans-differentiation in an influenza lung injury model (Liu et al., 2011), as well as advanced glycosylation end product-specific receptor (Ager), further supported a phenotypical switch at day 3 of culture (supplementary material Fig. S1). Here, AGER is linked to influenza.