Notably, downregulation of RORγt in group 3 ILCs is associated with the proliferation of these cells [28], and an increased frequency of group 3 ILCs expressed Ki67 following challenge with T. gondii. Therefore, the reduction in the group 3 ILC population during infection may be due a conversion of these cells into IFN-γ producing ILCs. Here, MKI67 is linked to infection.