Also, UGT1A9 exerts cancer prevention via inactivating benzo(α)pyrene (BaP) by glucuronidation [5]; UGT1A7*3 was regard as the sensitive polymorphism for carcinogens exposure due to its low catalytic activity [6]; metabolism of Diethylstilbestrol (DES) [7] and 4-(methylnitrosamino)-1-(3-pyridyl) -1-butanone (NNK) [8] conduct by UGT2B7 could influence chemical-induced carcinogenesis. This evidence concerns the gene UGT2B7 and cancer.