Since, LMTK2 was expressed in androgen dependent as well as androgen independent prostate cancer epithelial cells, it is likely that function of LMTK2 is not just limited to AR axis and certainly previous studies have shown LMTK2 to be an important binding partner of myosin VI involved in endocytic trafficking pathways in prostate cancer cells [33]. This evidence concerns the gene MYO6 and Familial prostate cancer.