At 24 hpi, in addition to the IFN response pathways, the pathogen pattern-recognition receptor (PRR) pathways (Toll-like receptor and RIG-I-like receptor signaling pathways), and inflammatory response related pathways (chemokine receptors binding chemokines and cytokine-cytokine receptor interaction) were also significantly enriched, suggesting that immune responses were greatly activated to combat viral infection. This evidence concerns the gene IFNA1 and viral infectious disease.