The greater association with the cardioembolic subtype emphasizes the role of a relationship between increased gravity and higher extent of cardioembolic stroke lesions23 and the activation of a subset of T cells such as the CD4+ and CD28− cells compared to the related component of the single plaque instability, than has been indicated by the few other studies that have addressed this issue.4–6. This evidence concerns the gene CD4 and cardioembolic stroke.