In a general linear model (R2 = 36 %) applied in polled diabetic groups, the differences between HNF1A–MODY and T1DM and between GCK–MODY and T1DM remained significant; their pattern and magnitude remained unchanged to that in univariate analysis (p < 0.001 for both comparisons). This evidence concerns the gene GCK and type 1 diabetes mellitus.