GATA1 and Dravet syndrome: DS-TMD, usually characterised by the presence of peripheral immature myeloblasts/megakaryoblasts and the variable involvement of other organs, is restricted to the neonatal period, spontaneously regresses and is the result of genetic co-operation between trisomy of HSA21 gene(s) with an acquired somatic mutation in GATA1 in virtually all cases [3].