We found that 96 hours after infection with Lv-Pro-miRNA126, the OX-LDL-induced apoptosis ratio was nearly the same in the apoptosis-resistant endothelial cells as that in normal endothelial cells, while BCL-2 gene silencing increased the apoptotic rate to approximately 94.2% in apoptosis-resistant endothelial cells (Fig 6C and 6D). This evidence concerns the gene BCL2 and infection.