Indeed, latent infection of Kasumi-3 cells results in all the hallmarks of latent infection observed in primary CD34+ cells, such as maintenance of the viral genome with concomitant lack of lytic IE72 expression but the presence of expression of the latency-associated LUNA gene (Albright & Kalejta, 2013; O'Connor & Murphy, 2012). The gene discussed is CD34; the disease is disease arising from reactivation of latent virus.