In support of this idea are: 1) the frequent aberrations in the AChE gene and the structural changes in AChE proteins observed in tumours of diverse origin [8, 15–19]; 2) the expression of AChE during and after apoptosis induction with different stimuli [20–22]; and 3) the profitable use as a prognostic predictor for liver carcinoma of AChE and its profitable effects through suppression of cell growth and enhancement of chemosensitization [23]. Here, ACHE is linked to neoplasm.