Given the data suggesting a role for Th1 and Th17 cells in mediating pain and inflammation in EAP and our previously published data demonstrating increased pain upon induction of pain by CP-1 infection in IFN-γKO mice [3], we next aimed to determine whether IL17 was important for either initiation or maintenance of prostatitis. The gene discussed is IL17A; the disease is urogenital neoplasm.