Several kinds of phenotypic abnormalities have been described in MDS such as a low SSC in granulocytic precursors, loss of antigen expression, asynchronous maturation or maturation block, aberrant cross-lineage co-expressions, quantitative and qualitative abnormalities of CD34+ cells, along with the decrease of precursor B cells (BCP) [9,11-21]. This evidence concerns the gene CD34 and myelodysplastic syndrome.