AKT1 and gastric cancer: AMP-18/peptide appears to act differently on p21 and cell homeostasis in non-transformed cells (Figs 1, 2D and 2E) compared to gastric cancer cells in which transfection of AMP-18 cDNA increased the level of p21, inhibited cell proliferation, induced apoptosis [61], and inactivated the PI3K/AKT pathway [62].