Those containing more genes were selected for the analysis (Fig 3B), which showed that more DE genes were clustered in the immune and inflammation response (cytokine–cytokine receptor interaction, MAPK signaling, Toll-like receptor (TLR) signaling, JAK-STAT signaling pathway, antigen processing and presentation, and Complement and coagulation cascades) and apoptosis in the H1N1-SS2 group than the two other infection groups. Here, SOAT1 is linked to infection.