In 1997, Koschinsky et al (7) showed that dietary MRPs partially absorbed into the bloodstream were chemically and biologically active, exerting harmful health effects, which is why they were called “glycotoxins.” This finding prompted extensive research confirming that consumption of large amounts of dietary MRPs might induce or aggravate insulin resistance, renal impairment or atherosclerosis, activate inflammatory and oxidative stress pathways, and contribute to development of complications in diabetes and nephropathies (8-11). The gene discussed is MRPS7; the disease is kidney disorder.