Although the LC5-RIC subclone shows a basal HERV-K (HML-2) transcription pattern that differs from that of the original LC5 as well as the parental LC5-RIC cells, infection with HIV-1 leads to a very similar HERV-K (HML-2) activation pattern (Table 3, Figure 6) compared to that of the infected parental LC5-RIC cells (Table 1, Figure 5). The gene discussed is CLEC10A; the disease is infection.