In the earlier studies, the exogenous protein of GFP and Cry1Ab/Ac could be digested without producing an allergic reaction [27, 28] and the TLR4 protein could be transferred to the endosome through the pathway Triad3A, Rab7b, and degenerated in the cytolysosome [29]. The gene discussed is RAB7B; the disease is allergic disease.