Integrating results with Oncomine data, we found miR-199a was cosuppressed or coexpressed with DNM2 mRNA; that is, significant underexpression of DNM2 (log2 median-centered intensity) was observed in HCC tumor (0.675) compared with precursor (0.717) and normal (0.870) liver tissues (Supplementary Figure S4), which is consistent with the underexpression of miR-199a in HCC tumor tissue. The gene discussed is DNM2; the disease is neoplasm.