Interestingly, in DOCA-salt hypertensive rats, Hernández et al. [22] found that the glucose, glycogen, and triglycerides levels were increased and citrate synthase and beta-hydroxyacyl-CoA dehydrogenase activities were reduced in the soleus muscle, along with higher plasma triglycerides and cholesterol compared with control rats; the study findings indicated that the hyperlipidemia of the DOCA-salt hypertensive animals was due to the changes of metabolic enzymes and not because of high food intake. The gene discussed is CS; the disease is hyperlipidemia.