Good syndrome (GS) is characterized as thymoma complicated with hypogammaglobulinemia and involves various immunodeficient conditions including depleted B cells, reduced T cells, and inversion of the CD4/CD8 ratio.4 It was first described by Dr. Robert Good in 1954.5 Although the pathogenesis of GS is still uncertain, a bone marrow defect impairing B-cell maturation has been suggested. This evidence concerns the gene CD8A and thymoma.