SLC2A2 and steatosis: Moreover, peroxisome proliferative activated receptor-γ co-activator 1 (PPARγ) expression, a stimulator of endogenous SLC2A2 mRNA transcription and key regulator of the genes associated with steatosis liver [12], was enhanced by about 40% after the leucine treatment (P ≤0.05; Figure 2A).