Along the same line, cardiomyocyte‐specific overexpression of eNOS improves left ventricular performance and reduces compensatory hypertrophy after MI.40 Therefore, the protective role of vitamin D, eNOS and NCC inhibitors in MI models underscores the possible importance of Fgf23 as a direct or indirect master regulator of all three factors. Here, SLC12A3 is linked to myocardial infarction.