Because infection with T. gondii is an important risk factor for developing schizophrenia, here we investigated how this infection may interact with the heterozygous deletion of the Nurr1 gene, which is necessary for dopamine neuron development, important for dopamine neurotransmission and also implicated to have a role in the development of schizophrenia [30–35]. This evidence concerns the gene NR4A2 and infection.