TGFB1 and diabetic kidney disease: Overexpression of miR-29 attenuates renal fibrosis in vivo in obstructive and diabetic nephropathies and suppresses the fibrotic genes in vitro in response to various stimuli including TGF-β1, high glucose or salt-induced hypertensive conditions (Du et al., 2010; Liu et al., 2010; Qin et al., 2011; Chen et al., 2014).