In the present study, we provide evidence for the first time that in patients with stable coronary artery disease and high levels of pro-atherogenic small dense LDL particles, monocyte subset distribution is skewed to a more "pro-inflammatory" profile with elevated levels of non-classical monocytes (CD14+CD16++) and reduced levels of classical monocytes (CD14++CD16-). Here, CD14 is linked to coronary artery disorder.