Similar mechanisms in the synovial membrane in RA and in the development of atherosclerotic plaques include T and B cells, macrophages, adhesive molecules, proinflammatory cytokines: tumor necrosis factor (TNF) alpha, interleukin 1 (IL-1), interleukin 6 (IL-6), and chemokines [12]. Here, IL6 is linked to rheumatoid arthritis.