In a study of adipogenic differentiation of mesenchymal stem cells (MSCs) from umbilical cord blood, genome-wide gene expression analysis identified significant (P = 3.03 x 10–4) upregulation of ADH1B during the change from undifferentiated to adipocyte-like differentiated MSCs.[61] The human chromosome 4q23 region has been shown to be homologous to a chromosomal region implicated in a murine model of abdominal obesity.[62] ADH1B is an adipogenic marker whose expression was limited by resveratrol-induced inhibition of clonal expansion and terminal differentiation of 3T3-L1 preadipocytes.[63]. Here, ADH1B is linked to Abdominal obesity.