IL-17A not only contributes to the epidermal abnormalities typical of psoriasis, but also induces the expression of chemokines in keratinocytes such as GRO-α (CXCL1) and IL-8 (CXCL8), which orchestrate the recruitment of neutrophils to psoriatic lesions 12. Here, CXCL1 is linked to psoriasis.