Because lipidation of LC3B is considered to be a primary event that is necessary for the induction of autophagy [39] and the level of conversion of LC3B-I to LC3B-II provides an indicator of autophagic activity, we examined levels of cytosolic LC3B-Iandlipidated LC3B-II after virus infection and isorhamnetin treatment. Here, MAP1LC3B is linked to viral infectious disease.