HuR is phosphorylated at tyrosine residues in tumor cells [27,28,29] and has several consensuses for phosphorylation by Abl-1, SRC, Lyn, FAK, and EGFR tyrosine kinases, involved in aberrant regulation of centrosomes in cancer cells [8,15,30,31,32,33]. This evidence concerns the gene ELAVL1 and neoplasm.