On the other hand, as intermittent human parathyroid hormone 1-34 (hPTH) exposure has been shown to enhance the formation of osteoblastic lesions in a mouse model of prostate cancer and this finding has been linked to elevated levels of bone turnover, the safety of other bone anabolic approaches needs to be assessed very carefully when used in this clinical setting [17]. The gene discussed is PTH; the disease is Familial prostate cancer.