CTRP3 replenishment following myocardial infarction has been shown to improve survival rates, to restore cardiac function, to attenuate cardiomyocyte apoptosis, to reduce fibrosis, and to increase revascularization, thus suggesting that reduced myocardial CTRP3 expression rather does not result in compensatory effects in the absence of adiponectin [29]. The gene discussed is ADIPOQ; the disease is myocardial infarction.