RGS1 and aortic aneurysm: Both of these chemokines are involved in monocyte recruitment to atherosclerotic lesions1, 23, 25 and their respective receptors are high on inflammatory Ly6Chi monocytes23, which were the cells recruited in our bead-tracking studies in aortic aneurysms, suggesting RGS1 acts as a ‘stop’ signal downstream of these receptors to reduce signalling and migration.