WAS and severe congenital neutropenia: A small subgroup of SCN patients have mutations in the transcriptional repressor oncoprotein growth factor independent (GFI) 1 [122], cytoskeletal regulator Wiskott-Aldrich Syndrome (WAS) protein [123], glucose-6 phosphatase catalytic subunit 3 (G6P3C) [124], endosomal adaptor protein p14 (also known as MAPBPIP) [125], or the endosomal trafficking vacuolar protein sorting 45 (VPS45) [126].