IGF1 and cervical adenocarcinoma: In the present study, we further observed that allosteric inhibition of mTORC1 by rapamycin suppressed IGF-1-stimulated cell adhesion in a panel of tumor cell lines, including human rhabdomyosarcoma (Rh30), Ewing sarcoma (Rh1), colon carcinoma (HT29), and cervical adenocarcinoma (HeLa) cells, which was not by reducing the cell viability.