It is reported that GC increased cell adhesion to the extracellular matrix via α-β1 integrin, and thereby antagonized epidermal growth factor (EGF) induced human gastric carcinoma cells migration [37]; MMF impaired transendothelial migration of allogeneic CD4 and CD8 T cells [38], inhibited intimal hyperplasia, and attenuates the expression of genes favoring smooth muscle cell proliferation and migration [39], while granulocyte precursors accumulate in the BM during mobilization induced by CYC leading to the accumulation of active neutrophil proteases [40]. Here, CD8A is linked to gastric carcinoma.