In the lipid system, the blood chemistry data showed that steroid hormone administration (model group) induced marked hyperlipidemia, such as significantly elevated TG (Figure 2(a)), TC (Figure 2(b)), LDL (Figure 2(c)), ApoA1 (Figure 2(e)), and ApoB (Figure 2(f)) levels, but significantly decreased HDL levels (Figure 2(d)). The gene discussed is APOB; the disease is hyperlipidemia.