Still in favor of IL-17 involvement in TLS induction, Peters et al. showed in a mouse model of multiple sclerosis [experimental autoimmune encephalomyelitis (EAE)] that the transfer of differentiated myelin oligodendrocyte glycoprotein (MOG)-specific Th17 cells was more capable of inducing ectopic lymphoid structures than other Th subtypes (96). The gene discussed is MOG; the disease is multiple sclerosis.