We have recently demonstrated that restoration of hCNT1 function in pancreatic adenocarcinoma cell lines is able to induce cell cycle arrest, increase cell death by a non-apoptotic mechanism, trigger changes in some intracellular signaling cascades and inhibit cell migration (Perez-Torras et al., 2013). This evidence concerns the gene SLC28A1 and pancreatic adenocarcinoma.