Given the fact that membrane-uncoupled p120 controls anchorage-independent tumor growth and metastasis of ILC (Schackmann et al., 2011), and nuclear p120 antagonizes Kaiso-dependent transcriptional repression in mILC cell lines, we hypothesized that Kaiso target genes could contribute to anchorage-independent survival of metastatic ILC. Here, CCL27 is linked to neoplasm.