Of the two wild-type TP53 cases, patient 12 had strong nuclear p53 protein accumulation consistent with p53 dysfunction, and patient 3 was reclassified as a synchronous HGSOC and high-grade uterine serous papillary carcinoma (based on simultaneous invasive uterine and ovarian lesions together with positive WT1 immunohistochemistry). This evidence concerns the gene TP53 and endometrial serous adenocarcinoma.